Abstract: Preeclampsia is a pregnancy-specific disorder that is not only a direct cause of adverse obstetric outcomes but also an independent risk factor for women’s long-term cardiovascular diseases, particularly coronary artery disease (CAD). This article systematically reviews the epidemiological evidence, risk characteristics, and mechanisms underlying the association between them. Studies have shown that preeclampsia advances the risk window for CAD. The underlying mechanisms involve the interaction and cascade effects of multiple pathways, including premature vascular aging, chronic inflammation, and metabolic disorders. The risk is particularly pronounced in women with early-onset preeclampsia, those complicated by adverse pregnancy outcomes (e.g., fetal growth restriction or placental abruption), and those with preconception traditional cardiovascular risk factors. Based on current evidence, the article proposes risk stratification and individualized intervention strategies, points out limitations in existing research related to population representativeness, biomarker-based prediction models, and targeted intervention trials, and suggests that future research should prioritize long-term studies and clinical integration tailored to the Chinese population.